SUMMARY
Achilles tendon rupture is the most common tendon rupture of the lower extremity, typically occurring after a sudden dorsiflexion force applied to a plantarflexed foot, most often during sporting activities.
Diagnosis is primarily clinical, characterized by weakness of plantarflexion and a positive Thompson test. Imaging is generally unnecessary in acute cases but may be helpful in chronic injuries or equivocal examinations, particularly when surgical planning is required.
Treatment options include nonoperative functional rehabilitation or operative repair, depending on patient age, activity level, comorbidities, and chronicity of the rupture.
EPIDEMIOLOGY
Incidence
Approximately 18 per 100,000 per year
Missed diagnosis reported in up to 20–25% of cases
Demographics
More common in men
Peak incidence between 30–40 years
Risk Factors
Episodic athletes (“weekend warriors”)
Fluoroquinolone antibiotic use
Corticosteroid injections
Prior intratendinous degeneration
Inflammatory arthropathies
ETIOLOGY
Mechanism of Injury
Acute traumatic injury, most commonly during sports
Sudden violent dorsiflexion of a plantarflexed foot
Less commonly with sudden forced plantarflexion
Pathoanatomy
Rupture typically occurs 4–6 cm proximal to the calcaneal insertion
This region corresponds to a hypovascular zone, predisposing to degeneration and rupture
ANATOMY
Achilles tendon is the largest and strongest tendon in the human body
Formed by the confluence of:
Soleus tendon
Medial gastrocnemius tendon
Lateral gastrocnemius tendon
Primary blood supply from the posterior tibial artery
Innervated via the tibial nerve
PRESENTATION
History
Sudden onset injury
Patients often describe a “pop” or snap
Frequently misdiagnosed as ankle sprain or DVT
Symptoms
Weakness with push-off
Difficulty walking
Posterior heel or calf pain
Physical Examination
Inspection
Increased resting ankle dorsiflexion with patient prone and knees flexed
Calf atrophy in chronic cases
Palpation
Palpable gap in the tendon (may be absent)
Motion
Weakness of ankle plantarflexion
Increased passive dorsiflexion
Provocative Tests
Thompson test
Squeezing the calf fails to produce plantarflexion
Strongly associated with complete rupture
· • Matles test (Knee Flexion Test)
o Prone position with knees flexed to 90°
o Increased resting ankle dorsiflexion compared with the contralateral side indicates Achilles tendon rupture
IMAGING
Radiographs
Indications
Rule out associated bony pathology
Not diagnostic for tendon rupture
Ultrasound
Indications
Differentiation between partial and complete rupture
Dynamic assessment
MRI
Indications
Equivocal physical exam
Chronic ruptures
Preoperative planning
Findings
Tendon discontinuity
Retraction of tendon ends
Degenerative changes in chronic cases
TREATMENT
Nonoperative Treatment
Method
Functional bracing or casting in resting equinus
Early functional rehabilitation preferred
Indications
Acute ruptures
Sedentary patients
Medically frail patients
Patient or surgeon preference
Outcomes
Equivalent plantarflexion strength compared to surgery
Level 1 evidence demonstrates equivalent re-rupture rates when functional rehab is used
Lower complication rates than operative treatment
Operative Treatment
Open End-to-End Achilles Tendon Repair
Indications
Acute ruptures (< 6 weeks)
High-demand or athletic patients
Outcomes
Historically lower re-rupture rates
No significant difference in strength or re-rupture rates compared to nonoperative treatment when functional rehab protocols are used
Increased risk of wound complications
Percutaneous Achilles Tendon Repair
Indications
Desire to minimize wound complications
Cosmetic concerns
Outcomes
Higher risk of sural nerve injury
Lower wound complication rates
Faster return to work
Equivalent functional outcomes at 1 year
Reconstruction with V–Y Advancement
Indications
Chronic ruptures with tendon defect < 3 cm
Flexor Hallucis Longus (FHL) Transfer ± V–Y Advancement
Indications
Chronic ruptures with defect > 3 cm
Requires an intact tibial nerve
Considerations
Possible residual hallux plantarflexion weakness
TECHNIQUES
Functional Bracing / Casting
Immobilization at ~20° plantarflexion
Early functional rehabilitation encouraged
Weeks 0–2
• Immobilization in a cast or functional boot at approximately 20° plantarflexion
• Non–weight bearing or protected touch-down weight bearing with crutches
• Emphasis on:
pain control
edema reduction
• No active ankle range of motion
Weeks 2–4
• Transition to a functional walking boot with heel wedges maintaining plantarflexion
• Initiate protected weight bearing as tolerated
• Begin gentle active plantarflexion within the boot
• Avoid dorsiflexion beyond neutral
• No stretching of the Achilles tendon
Weeks 4–6
• Gradual removal of heel wedges to allow progressive dorsiflexion toward neutral
• Progress to full weight bearing in the boot
• Initiate:
active ankle range of motion (plantarflexion to neutral)
gentle isometric strengthening
• Continue to avoid forced dorsiflexion
Weeks 6–8
• Discontinue boot as tolerated and transition to supportive footwear with heel lift
• Begin:
active dorsiflexion beyond neutral
light concentric strengthening of the gastrocnemius–soleus complex
• Gait training to normalize walking pattern
Weeks 8–12
• Progress strengthening exercises:
heel raises (bilateral → unilateral)
proprioceptive and balance training
• Gradual return to low-impact activities
• Avoid explosive push-off and running
After 12 Weeks
• Advance to:
sport-specific training
plyometric exercises as tolerated
• Gradual return to running and high-impact activities
• Full return to sport typically expected at 4–6 months
Open Repair
Medial incision to avoid sural nerve
Paratenon incised
Repair with heavy non-absorbable sutures
Postoperative immobilization in plantarflexion for 4–6 weeks
Percutaneous Repair
Multiple small stab incisions
Careful protection of sural nerve
V–Y Advancement
V-shaped incision at musculotendinous junction
Superficial tendon lengthened while preserving muscle fibers
FHL Transfer
Degenerative tendon excised
FHL harvested at Knot of Henry
Tendon transferred through calcaneus
COMPLICATIONS
Re-rupture
Historically higher in nonoperative treatment
No difference with modern functional rehabilitation
Treated with surgical repair
Wound Complications
Incidence: 5–10%
Risk factors:
Smoking (most significant)
Steroid use
Female sex
Open surgical technique
Infection
Managed with aggressive debridement
Culture-specific antibiotics for 6 weeks
Sural Nerve Injury
More common with percutaneous techniques