DIABETIC NEUROPATHY AND CHARCOT NEUROARTHROPATHY

MD Murat BIRINCI· Umraniye Training and Research Hospital, Department of Ortopaedics and Traumatology, Istanbul
Apr 22, 2026

SUMMARY

Diabetic Charcot Neuroarthropathy is a chronic, progressive, and destructive condition resulting from loss of protective sensation, leading to collapse and deformity of the foot and ankle joints.
Clinically, it presents with a warm, swollen, erythematous foot, often mistaken for infection; however, erythema characteristically decreases with elevation.
Radiographs demonstrate joint space obliteration, osseous fragmentation, and joint subluxation or dislocation.
Initial management consists of strict immobilization with total contact casting in acute stages. Surgical intervention is reserved for severe, unstable, or non-braceable deformities.


EPIDEMIOLOGY

Incidence

• 0.1–1.4% of patients with diabetes
• Up to 7.5% of patients with diabetes and established neuropathy

Demographics

Type 1 diabetes: typically presents in the 5th decade (20–25 years after diagnosis)
Type 2 diabetes: typically presents in the 6th decade (5–10 years after diagnosis)

Anatomic Distribution

• Foot and ankle (most common)
• Bilateral involvement in 9–35%
• Less commonly: knee, shoulder, elbow


ETIOLOGY AND PATHOPHYSIOLOGY

Peripheral Diabetic Neuropathy

• Sensory neuropathy → loss of protective sensation
• Motor neuropathy → muscle imbalance and progressive deformity
• Autonomic neuropathy → increased local blood flow and bone resorption

Pathophysiologic Theories

Neurotraumatic theory
• Insensate joints subjected to repetitive microtrauma
• Inability to mount protective responses

Neurovascular theory
• Autonomic dysfunction → arteriovenous shunting
• Increased bone turnover and weakening

Molecular Mechanisms

• Elevated inflammatory cytokines (IL-1, TNF-α)
• Activation of NF-κB
• Dysregulation of RANK / RANKL / OPG pathway → osteolysis


CLASSIFICATION

Brodsky Classification (Anatomic)

Type 1: Tarsometatarsal & naviculocuneiform joints (60%)
 – Midfoot collapse → rocker-bottom deformity

Type 2: Subtalar, talonavicular, calcaneocuboid joints (10%)
 – Highly unstable, prolonged immobilization required

Type 3A: Tibiotalar joint (20%)
 – Progressive varus or valgus deformity

Type 3B: Calcaneal tuberosity fracture (<10%)

Type 4: Combined regions (<10%)

Type 5: Forefoot only (<10%)


Eichenholtz Classification (Temporal)

Stage 0 (Prefragmentation):
 – Clinical swelling and warmth, radiographs normal

Stage 1 (Fragmentation):
 – Osseous fragmentation, joint dislocation

Stage 2 (Coalescence):
 – Decreased edema, fragment resorption

Stage 3 (Reconstruction):
 – Consolidation and remodeling


PRESENTATION

Symptoms

• Swelling and warmth
• Pain present in ~50%, painless in others
• Functional instability

Physical Examination

Acute Charcot
• Diffuse swelling
• Temperature difference ≥3°C compared to contralateral side
• Erythema that decreases with elevation

Chronic Charcot
• Structural deformity
• Bony prominences
• Rocker-bottom foot
• Ligamentous instability

Neurologic Exam
• Semmes-Weinstein 5.07 monofilament
• Sensitivity: 40–95%


IMAGING

Radiographs

• Weight-bearing AP and lateral foot views
• Ankle series

Findings
• Early: degenerative changes mimicking osteoarthritis
• Late:

  • Joint space obliteration

  • Osseous fragmentation

  • Subluxation or dislocation

  • Heterotopic ossification

MRI

• Helpful for soft tissue evaluation
• Limited specificity in distinguishing Charcot from infection


TREATMENT

Nonoperative (First-Line)

Total Contact Casting

  • Immobilization with strict non–weight bearing

  • Cast changes every 2–4 weeks

  • Duration: 2–4 months

Transition Bracing

  • Charcot Restraint Orthotic Walker (CROW)

Footwear Modifications

  • Double rocker sole in Eichenholtz stage 3

Medical Management

  • Bisphosphonates

  • Neuropathic pain agents

Success rate: ~75%


Operative

Indications
• Non-braceable deformity
• Recurrent instability
• Severe structural collapse

Procedures
• Exostectomy ± TAL for focal deformities
• Deformity correction with arthrodesis
• External fixation when bone quality is poor

Outcomes
• Complication rates up to 70%
• High risk of infection, nonunion, and hardware failure


PROGNOSIS

Depends on:
• Stage at diagnosis
• Degree of deformity
• Patient compliance with immobilization
• Neuropathy severity